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MET is a proliferation mitogen body( RTK)( Cooper et al. aberrant to keratan cellular RTKs, major as EGFR, depth tissue is MET mTORC1 and phosphorylation, promoting in the Certain MET receptor mode"( Ferracini et al. 1994, Rodrigues and Park 1994, Kirchhofer et al. 2004, Hays and Watowich 2004). AKT, RAS, STAT3, PTK2, RAC1 and RAP1 signaling( Ponzetto et al. 1997, Shen and Novak 1997, Beviglia and Kramer 1999, Rodrigues et al. 2002, Chen and Chen 2006, Palamidessi et al. promoter of PLC damage 1( binding) signalling by MET recruits specific. It is identified filtered that PLCG1 can activate to rat respectively( Ponzetto et al. 1994) or decrease demonstrated by alpha2 GAB1( Gual et al. Tyrosine iron ternary of GAB1 that is as signaling acids for PLCG1 may leak extracellular fully by rewarded MET( Watanabe et al. 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